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Vivitrol / ReVia Tapering Guide

naltrexone

OtherFDA 1984
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Overview

Naltrexone is an opioid antagonist used for alcohol use disorder and opioid use disorder relapse prevention. Low-dose naltrexone (4.5mg, LDN) is used off-label for chronic pain, fibromyalgia, and inflammatory conditions — different mechanism, different evidence base.

Common Doses

50mg tablets (oral); 380mg IM monthly (Vivitrol); 4.5mg compounded (low-dose naltrexone, off-label)

Formulations

Tablets: 50mg; IM (Vivitrol): 380mg/month; Compounded LDN: 1.5-4.5mg (off-label)

Pregnancy

Category C

Mechanism of Action

Competitive antagonist at mu, kappa, and delta opioid receptors. At low doses, paradoxical effects may involve transient receptor blockade and endogenous opioid upregulation.

Taper Notes

For oral naltrexone, most patients can stop without taper. For depot, the long half-life provides a natural taper. Plan for return of cravings.

Hyperbolic Tapering Guidance

Naltrexone is not dependence-forming; the main risk on discontinuation is loss of the medication-supported buffer against the underlying disorder, not pharmacological withdrawal.

Summary written by TaperCommunity, informed by the Maudsley Deprescribing Guidelines (Horowitz & Taylor) and related literature — see Sources & References below. Not affiliated with or endorsed by the Maudsley.

Withdrawal Timeline

Onset

Days (oral) or weeks (depot wear-off)

📈Peak Severity

1-2 weeks

📉Resolution

Usually 2-4 weeks

⚠️Protracted Risk

Rare for the medication itself; the underlying condition is the main long-term consideration

Common Withdrawal Symptoms

mild — naltrexone itself does not produce dependenceunderlying alcohol/opioid cravings can return without the medication-supported buffer

Interactions & Safety

Drug Interactions

  • All opioids — naltrexone blocks effects; if opioid is needed for pain, will require much higher doses with careful monitoring
  • Yohimbine — additive effects

Contraindications

  • Current opioid use or dependence (will precipitate withdrawal)
  • Acute opioid withdrawal
  • Severe hepatic impairment

Toxicity

Hepatotoxicity (dose-dependent — uncommon at standard doses), nausea, headache, dizziness, anxiety, insomnia, depression. Will precipitate severe withdrawal in opioid-dependent patients.

Pharmacokinetics

ADME Profile

Metabolism

Hepatic via dihydrodiol dehydrogenase.

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Sources & References

Vivitrol / ReVia (naltrexone) information on this page is sourced from peer-reviewed research, regulatory bodies, clinical guidelines, and patient-advocacy organizations.

Encyclopedic & chemical databases

Neutral, high-authority entity references.

Regulatory sources

Official prescribing information and safety notices.

Deprescribing-specific resources

Clinician-facing references on tapering protocols.

Patient-advocacy & lived-experience

Long-running communities documenting withdrawal experience.

TaperCommunity does not provide medical advice. Always consult a qualified prescriber before adjusting psychiatric medication.